Homozygotes for targeted null mutations exhibit cardiac hypertrophy, ventricular septal defects, pulmonary and liver congestion, and reduced preweaning viability.
|Process||anterior/posterior axis specification, arterial endothelial cell differentiation, ...|
|Component||cytoplasm, nucleus, ...|
|Function||DNA binding, histone deacetylase binding, ...|
|RNA in situ||333|
|In situ reporter (knock in)||14|
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Tumor Biology (MTB), Gene Ontology (GO), MouseCyc
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