About   Help   FAQ
Phenotypes associated with this allele
Allele Symbol
Allele Name
Allele ID
Tcra-Jtm1Tgi
targeted mutation 1, Masaru Taniguchi
MGI:2180736
Summary 9 genotypes
Jump to Allelic Composition Genetic Background Genotype ID
hm1
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi B6.129-Tcra-Jtm1Tgi MGI:3722107
hm2
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi C.129-Tcra-Jtm1Tgi MGI:5426352
hm3
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi involves: 129S1/Sv * 129X1/SvJ MGI:3813929
hm4
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi involves: 129S1/Sv * 129X1/SvJ * BALB/c MGI:4839084
hm5
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi involves: 129S1/Sv * 129X1/SvJ * C57BL/6 MGI:4839085
cx6
Ldlrtm1Her/Ldlrtm1Her
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
B6.129-Ldlrtm1Her Tcra-Jtm1Tgi MGI:4839086
cx7
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Tg(Pklr-Myc)73Ak/0
B6.Cg-Tcra-Jtm1Tgi Tg(Pklr-Myc)73Ak MGI:5430591
cx8
Sh2d1atm1Cpt/Sh2d1atm1Cpt
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
involves: 129S1/Sv * 129S4/SvJae * 129X1/SvJ * BALB/c MGI:5444640
cx9
Sh2d1atm1Cpt/Y
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
involves: 129S1/Sv * 129S4/SvJae * 129X1/SvJ * BALB/c MGI:5444644


Genotype
MGI:3722107
hm1
Allelic
Composition
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
B6.129-Tcra-Jtm1Tgi
Find Mice Using the International Mouse Strain Resource (IMSR)
No mouse lines available in IMSR.
See publication links below for author information.
phenotype observed in females
phenotype observed in males
N normal phenotype
immune system
• the numbers of CD69+ (a marker for activation) B cells in the spleen are reduced (4.1+/-0.5 compared to 5.1+/-0.7 in wild-type mice) (J:99107)
• the numbers of CD69+ (a marker for activation) B cells in the spleen are reduced (4.1+/-0.5 compared to 5.1+/-0.7 in wild-type mice) (J:99107)
• the numbers of CD69+ (a marker for activation) T cells in the spleen are reduced (9.9+/-1.8 compared to 16.0+/-3.4 in wild-type mice) (J:99107)
• the numbers of CD69+ (a marker for activation) T cells in the spleen are reduced (9.9+/-1.8 compared to 16.0+/-3.4 in wild-type mice) (J:99107)
• anti OVA IgE levels are about one-eighth of control levels after challenge (J:121037)
• anti OVA IgE levels are about one-eighth of control levels after challenge (J:121037)
• levels of anti CII IgG antibodies are reduced compared to in wild-type mice (J:99107)
• levels of anti CII IgG antibodies are reduced compared to in wild-type mice (J:99107)
• 24 hours after OVA challenge (J:121037)
• 24 hours after OVA challenge (J:121037)
• following induction of arthritis with collagen (CIA) the incidence (40%) and arthritis score (1.5+/-2.2) is reduced compared to wild-type mice (90% incidence with a score of 5.4+/-3.2) (J:99107)
• following induction of arthritis with collagen (CIA) the incidence (40%) and arthritis score (1.5+/-2.2) is reduced compared to wild-type mice (90% incidence with a score of 5.4+/-3.2) (J:99107)
• marked infiltration of neutrophiles at 24 hours after cauterization (J:132503)
• no further neutrophil or macrophage increases to 96 hours (J:132503)
• increased edema at 96 hours after cauterization (J:132503)
• marked infiltration of neutrophiles at 24 hours after cauterization (J:132503)
• no further neutrophil or macrophage increases to 96 hours (J:132503)
• increased edema at 96 hours after cauterization (J:132503)
• after BDL, necroinflammatory foci and lymphocytic infiltration are obviously less than in controls (J:135830)
• after BDL, necroinflammatory foci and lymphocytic infiltration are obviously less than in controls (J:135830)
• 42% fewer cells in bronchoalveolar lavage fluid compared to controls after OVA sensitization and challenge (J:121037)
• eosinophiles reduced by 58% (J:121037)
• reduced macrophage and neutrophiles (J:121037)
• normal lymphocyte recruitment (J:121037)
• IL-4 and IL-5 also reduced in bronchoalveolar lavage fluid 24 hours after challenge (J:121037)
• 42% fewer cells in bronchoalveolar lavage fluid compared to controls after OVA sensitization and challenge (J:121037)
• eosinophiles reduced by 58% (J:121037)
• reduced macrophage and neutrophiles (J:121037)
• normal lymphocyte recruitment (J:121037)
• IL-4 and IL-5 also reduced in bronchoalveolar lavage fluid 24 hours after challenge (J:121037)

homeostasis/metabolism
• 24 hours after OVA challenge (J:121037)
• 24 hours after OVA challenge (J:121037)
• one day following bile duct ligation (BDL), serum ALT levels are significantly lower than controls (J:135830)
• one day following bile duct ligation (BDL), serum ALT levels are significantly lower than controls (J:135830)

liver/biliary system
• hepatocyte cell death is reduced compared to controls after BDL (J:135830)
• hepatocyte cell death is reduced compared to controls after BDL (J:135830)
• after BDL, necroinflammatory foci and lymphocytic infiltration are obviously less than in controls (J:135830)
• after BDL, necroinflammatory foci and lymphocytic infiltration are obviously less than in controls (J:135830)
• confluent foci of feathery hepatocyte degeneration due to bile acid cytotoxicity are significantly reduced compared to controls 24 hours after BDL (J:135830)
• confluent foci of feathery hepatocyte degeneration due to bile acid cytotoxicity are significantly reduced compared to controls 24 hours after BDL (J:135830)
• necroinflammatory foci after BDL are reduced in number compared to controls (J:135830)
• necroinflammatory foci after BDL are reduced in number compared to controls (J:135830)

hematopoietic system
• the numbers of CD69+ (a marker for activation) B cells in the spleen are reduced (4.1+/-0.5 compared to 5.1+/-0.7 in wild-type mice) (J:99107)
• the numbers of CD69+ (a marker for activation) B cells in the spleen are reduced (4.1+/-0.5 compared to 5.1+/-0.7 in wild-type mice) (J:99107)
• the numbers of CD69+ (a marker for activation) T cells in the spleen are reduced (9.9+/-1.8 compared to 16.0+/-3.4 in wild-type mice) (J:99107)
• the numbers of CD69+ (a marker for activation) T cells in the spleen are reduced (9.9+/-1.8 compared to 16.0+/-3.4 in wild-type mice) (J:99107)
• anti OVA IgE levels are about one-eighth of control levels after challenge (J:121037)
• anti OVA IgE levels are about one-eighth of control levels after challenge (J:121037)
• levels of anti CII IgG antibodies are reduced compared to in wild-type mice (J:99107)
• levels of anti CII IgG antibodies are reduced compared to in wild-type mice (J:99107)

skeleton
• following induction of arthritis with collagen (CIA) the incidence (40%) and arthritis score (1.5+/-2.2) is reduced compared to wild-type mice (90% incidence with a score of 5.4+/-3.2) (J:99107)
• following induction of arthritis with collagen (CIA) the incidence (40%) and arthritis score (1.5+/-2.2) is reduced compared to wild-type mice (90% incidence with a score of 5.4+/-3.2) (J:99107)

cardiovascular system
• area of choroidal neovascularization stimulated by photocoagulation is significantly reduced (J:138197)
• area of choroidal neovascularization stimulated by photocoagulation is significantly reduced (J:138197)

vision/eye
• area of choroidal neovascularization stimulated by photocoagulation is significantly reduced (J:138197)
• area of choroidal neovascularization stimulated by photocoagulation is significantly reduced (J:138197)
• marked infiltration of neutrophiles at 24 hours after cauterization (J:132503)
• no further neutrophil or macrophage increases to 96 hours (J:132503)
• increased edema at 96 hours after cauterization (J:132503)
• marked infiltration of neutrophiles at 24 hours after cauterization (J:132503)
• no further neutrophil or macrophage increases to 96 hours (J:132503)
• increased edema at 96 hours after cauterization (J:132503)
• cauterized area of cornea becomes thickened (J:132503)
• cauterized area of cornea becomes thickened (J:132503)
• increased opacity relative to controls 24 hours after corneal cauterization (J:132503)
• opacity increases further by 96 hours (J:132503)
• increased opacity relative to controls 24 hours after corneal cauterization (J:132503)
• opacity increases further by 96 hours (J:132503)

reproductive system
• alpha-galactosylceramide induced abortion does not occur (J:59892)
• alpha-galactosylceramide induced abortion does not occur (J:59892)

respiratory system
• 42% fewer cells in bronchoalveolar lavage fluid compared to controls after OVA sensitization and challenge (J:121037)
• eosinophiles reduced by 58% (J:121037)
• reduced macrophage and neutrophiles (J:121037)
• normal lymphocyte recruitment (J:121037)
• IL-4 and IL-5 also reduced in bronchoalveolar lavage fluid 24 hours after challenge (J:121037)
• 42% fewer cells in bronchoalveolar lavage fluid compared to controls after OVA sensitization and challenge (J:121037)
• eosinophiles reduced by 58% (J:121037)
• reduced macrophage and neutrophiles (J:121037)
• normal lymphocyte recruitment (J:121037)
• IL-4 and IL-5 also reduced in bronchoalveolar lavage fluid 24 hours after challenge (J:121037)
• less airway hypersensitive response develops after OVA sensitization and challenge (J:121037)
• less airway hypersensitive response develops after OVA sensitization and challenge (J:121037)

cellular
• hepatocyte cell death is reduced compared to controls after BDL (J:135830)
• hepatocyte cell death is reduced compared to controls after BDL (J:135830)




Genotype
MGI:5426352
hm2
Allelic
Composition
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
C.129-Tcra-Jtm1Tgi
Find Mice Using the International Mouse Strain Resource (IMSR)
No mouse lines available in IMSR.
See publication links below for author information.
phenotype observed in females
phenotype observed in males
N normal phenotype

Il17rbtm1Hwata/Il17rbtm1Hwata and Tcra-Jtm1Tgi/Tcra-Jtm1Tgi mice fail to exhibit airway hyperactivity in response to respiratory syncytial virus exposure

immune system
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus fail to exhibit an increase in IL5, IL9, IL10, IL13, IL17A and IL22 unlike wild-type mice (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus fail to exhibit an increase in IL5, IL9, IL10, IL13, IL17A and IL22 unlike wild-type mice (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)
• the bronchoalveolar lavage fluid from mice exposed to respiratory syncytial virus (J:184570)

respiratory system
• in response to respiratory syncytial virus exposure, mice fail to exhibit airway hyperreactivity unlike wild-type mice (J:184570)
• however, the response could be restored by transfer of wild-type invariant NK T cells (J:184570)
• in response to respiratory syncytial virus exposure, mice fail to exhibit airway hyperreactivity unlike wild-type mice (J:184570)
• however, the response could be restored by transfer of wild-type invariant NK T cells (J:184570)




Genotype
MGI:3813929
hm3
Allelic
Composition
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
involves: 129S1/Sv * 129X1/SvJ
Find Mice Using the International Mouse Strain Resource (IMSR)
No mouse lines available in IMSR.
See publication links below for author information.
phenotype observed in females
phenotype observed in males
N normal phenotype
cardiovascular system
N
• mice exhibit normal arteriogenesis following occlusion (J:134875)
• mice exhibit normal arteriogenesis following occlusion (J:134875)

immune system
• IFN gamma is not induced by LPS administration in Propionibacterium acnes primed mice (J:155199)
• IFN gamma is not induced by LPS administration in Propionibacterium acnes primed mice (J:155199)
• decreased production by CD4 T-cells after exposure to heat killed Propionibacterium acnes (J:155199)
• decreased production by CD4 T-cells after exposure to heat killed Propionibacterium acnes (J:155199)
• expression data indicates absence of liver damage in response to LPS administration in Propionibacterium acnes primed (J:155199)
• expression data indicates absence of liver damage in response to LPS administration in Propionibacterium acnes primed (J:155199)

homeostasis/metabolism
• IFN gamma is not induced by LPS administration in Propionibacterium acnes primed mice (J:155199)
• IFN gamma is not induced by LPS administration in Propionibacterium acnes primed mice (J:155199)




Genotype
MGI:4839084
hm4
Allelic
Composition
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
involves: 129S1/Sv * 129X1/SvJ * BALB/c
Find Mice Using the International Mouse Strain Resource (IMSR)
No mouse lines available in IMSR.
See publication links below for author information.
phenotype observed in females
phenotype observed in males
N normal phenotype
respiratory system
• hyper-reactivity does not develop in response to OVA challenge (J:84720)
• hyper-reactivity does not develop in response to OVA challenge (J:84720)

immune system
• substantial reduction in airway eosinophilia in response to OVA challenge (J:84720)
• substantial reduction in airway eosinophilia in response to OVA challenge (J:84720)
• decreased OVA specific IgE in serum of OVA challenged mice (J:84720)
• decreased OVA specific IgE in serum of OVA challenged mice (J:84720)
• less IL-13 found in bronchial lymph nodes of OVA challenged mice (J:84720)
• less IL-13 found in bronchial lymph nodes of OVA challenged mice (J:84720)
• less IL-4 found in bronchial lymph nodes of OVA challenged mice (J:84720)
• less IL-4 found in bronchial lymph nodes of OVA challenged mice (J:84720)
• resistant to the IL-12 induced increase in mononuclear cells after partial hepatectomy seen in controls (J:105977)
• resistant to the IL-12 induced increase in mononuclear cells after partial hepatectomy seen in controls (J:105977)

liver/biliary system
• resistant to the IL-12 induced increase in mononuclear cells after partial hepatectomy seen in controls (J:105977)
• resistant to the IL-12 induced increase in mononuclear cells after partial hepatectomy seen in controls (J:105977)
• IL-12 fails to cause any liver damage beyond that caused by partial hepatectomy (J:105977)
• IL-12 fails to cause any liver damage beyond that caused by partial hepatectomy (J:105977)

hematopoietic system
• substantial reduction in airway eosinophilia in response to OVA challenge (J:84720)
• substantial reduction in airway eosinophilia in response to OVA challenge (J:84720)
• decreased OVA specific IgE in serum of OVA challenged mice (J:84720)
• decreased OVA specific IgE in serum of OVA challenged mice (J:84720)




Genotype
MGI:4839085
hm5
Allelic
Composition
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
involves: 129S1/Sv * 129X1/SvJ * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
No mouse lines available in IMSR.
See publication links below for author information.
phenotype observed in females
phenotype observed in males
N normal phenotype
mortality/aging
• alpha-galactosylceramide has no protective effect against bleomycin induced mortality (J:89695)
• 65% die by the 28th day (J:89695)
• alpha-galactosylceramide has no protective effect against bleomycin induced mortality (J:89695)
• 65% die by the 28th day (J:89695)

respiratory system
• initial development of pulmonary inflammation develops after bleomycin administration in mice treated with alpha-galactosylceramide just as in controls (J:89695)
• initial development of pulmonary inflammation develops after bleomycin administration in mice treated with alpha-galactosylceramide just as in controls (J:89695)
• develops after bleomycin administration in mice treated with alpha-galactosylceramide just as in controls (J:89695)
• develops after bleomycin administration in mice treated with alpha-galactosylceramide just as in controls (J:89695)

hematopoietic system
• no NKT cells are found in the lungs after bleomycin administration (J:89695)
• no NKT cells are found in the lungs after bleomycin administration (J:89695)

liver/biliary system
• considerably less liver damage induced by concanavalin A injection than in controls (J:93915)
• as measured by alanine transaminase levels (J:93915)
• reduced concanavalin A induced histological damage (J:93915)
• considerably less liver damage induced by concanavalin A injection than in controls (J:93915)
• as measured by alanine transaminase levels (J:93915)
• reduced concanavalin A induced histological damage (J:93915)

homeostasis/metabolism
• alpha-galactosylceramide has no protective effect against bleomycin induced mortality (J:89695)
• 65% die by the 28th day (J:89695)
• alpha-galactosylceramide has no protective effect against bleomycin induced mortality (J:89695)
• 65% die by the 28th day (J:89695)
• considerably less liver damage induced by concanavalin A injection than in controls (J:93915)
• as measured by alanine transaminase levels (J:93915)
• reduced concanavalin A induced histological damage (J:93915)
• considerably less liver damage induced by concanavalin A injection than in controls (J:93915)
• as measured by alanine transaminase levels (J:93915)
• reduced concanavalin A induced histological damage (J:93915)

immune system
• no NKT cells are found in the lungs after bleomycin administration (J:89695)
• no NKT cells are found in the lungs after bleomycin administration (J:89695)
• initial development of pulmonary inflammation develops after bleomycin administration in mice treated with alpha-galactosylceramide just as in controls (J:89695)
• initial development of pulmonary inflammation develops after bleomycin administration in mice treated with alpha-galactosylceramide just as in controls (J:89695)




Genotype
MGI:4839086
cx6
Allelic
Composition
Ldlrtm1Her/Ldlrtm1Her
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
B6.129-Ldlrtm1Her Tcra-Jtm1Tgi
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Ldlrtm1Her mutation (18 available); any Ldlr mutation (46 available)
Tcra-Jtm1Tgi mutation (0 available); any Tcra-J mutation (0 available)
phenotype observed in females
phenotype observed in males
N normal phenotype
cardiovascular system
• lesions in the ascending aorta are reduced 20% in males and 28% in females relative to homozygous Ldlrtm1Her (J:140276)
• considerable reduction of lipid containing areas in the lesions (J:140276)
• less IFN-gamma in the lesions (J:140276)
• lesions in the ascending aorta are reduced 20% in males and 28% in females relative to homozygous Ldlrtm1Her (J:140276)
• considerable reduction of lipid containing areas in the lesions (J:140276)
• less IFN-gamma in the lesions (J:140276)

immune system
• less IFN-gamma in atherosclerotic lesions (J:140276)
• less IFN-gamma in atherosclerotic lesions (J:140276)




Genotype
MGI:5430591
cx7
Allelic
Composition
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Tg(Pklr-Myc)73Ak/0
Genetic
Background
B6.Cg-Tcra-Jtm1Tgi Tg(Pklr-Myc)73Ak
Find Mice Using the International Mouse Strain Resource (IMSR)
No mouse lines available in IMSR.
See publication links below for author information.
phenotype observed in females
phenotype observed in males
N normal phenotype
tumorigenesis
• 40% of double mutants exhibit lung metastasis which is not seen in single Tg(Pklr-Myc)73Ak mice (J:184496)
• 40% of double mutants exhibit lung metastasis which is not seen in single Tg(Pklr-Myc)73Ak mice (J:184496)
• tumors have higher grades of malignancy and are less differentiated than those of single Tg(Pklr-Myc)73Ak mice (J:184496)
• tumors have higher grades of malignancy and are less differentiated than those of single Tg(Pklr-Myc)73Ak mice (J:184496)
• mutants develop hepatocellular carcinoma (HCC); stronger promotion of initiation and progression steps of HCC development compared to single Tg(Pklr-Myc)73Ak mice (J:184496)
• the number and size of tumor nodules in double mutants is greater than in single Tg(Pklr-Myc)73Ak mice (J:184496)
• some tumors in double mutants exhibit a very poorly differentiated phenotype (fetal-like phenotype) that is never seen in tumors of single Tg(Pklr-Myc)73Ak mice (J:184496)
• tumors have higher mitotic indices compared with tumors from single Tg(Pklr-Myc)73Ak mice (J:184496)
• mutants develop hepatocellular carcinoma (HCC); stronger promotion of initiation and progression steps of HCC development compared to single Tg(Pklr-Myc)73Ak mice (J:184496)
• the number and size of tumor nodules in double mutants is greater than in single Tg(Pklr-Myc)73Ak mice (J:184496)
• some tumors in double mutants exhibit a very poorly differentiated phenotype (fetal-like phenotype) that is never seen in tumors of single Tg(Pklr-Myc)73Ak mice (J:184496)
• tumors have higher mitotic indices compared with tumors from single Tg(Pklr-Myc)73Ak mice (J:184496)

Mouse Models of Human Disease
OMIM ID Ref(s)
Hepatocellular Carcinoma 114550 J:184496




Genotype
MGI:5444640
cx8
Allelic
Composition
Sh2d1atm1Cpt/Sh2d1atm1Cpt
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
involves: 129S1/Sv * 129S4/SvJae * 129X1/SvJ * BALB/c
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Sh2d1atm1Cpt mutation (0 available); any Sh2d1a mutation (3 available)
Tcra-Jtm1Tgi mutation (0 available); any Tcra-J mutation (0 available)
phenotype observed in females
phenotype observed in males
N normal phenotype
immune system
• following immunization with NP-KLH (J:189126)
• following immunization with NP-KLH (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)

hematopoietic system
• following immunization with NP-KLH (J:189126)
• following immunization with NP-KLH (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)




Genotype
MGI:5444644
cx9
Allelic
Composition
Sh2d1atm1Cpt/Y
Tcra-Jtm1Tgi/Tcra-Jtm1Tgi
Genetic
Background
involves: 129S1/Sv * 129S4/SvJae * 129X1/SvJ * BALB/c
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Sh2d1atm1Cpt mutation (0 available); any Sh2d1a mutation (3 available)
Tcra-Jtm1Tgi mutation (0 available); any Tcra-J mutation (0 available)
phenotype observed in females
phenotype observed in males
N normal phenotype
hematopoietic system
• following immunization with NP-KLH (J:189126)
• following immunization with NP-KLH (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)

immune system
• following immunization with NP-KLH (J:189126)
• following immunization with NP-KLH (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)
• of NP-specific IgG following immunization with NP-KLH that is more severe than in Sh2d1atm1Cpt homozygotes on a similar background (J:189126)





Contributing Projects:
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Tumor Biology (MTB), Gene Ontology (GO), MouseCyc
Citing These Resources
Funding Information
Warranty Disclaimer & Copyright Notice
Send questions and comments to User Support.
last database update
02/02/2016
MGI 6.02
The Jackson Laboratory