Mice homozygous for disruptions in this gene are fertile and viable with no obvious phenotypic abnormality. Cellular responses of macrophages to extracellular ATP are frequently normal however. In addition, long bones are thinner than normal in adult mice.
|VEGA Gene Model||OTTMUSG00000026740 (Evidence)|
|Ensembl Gene Model||ENSMUSG00000029468 (Evidence)|
|Entrez Gene||18439 (Evidence)|
|DFCI||TC1588530, TC1585870, TC1625377, TC1589640|
|DoTS||DT.94273607, DT.99849462, DT.94391133, DT.101355105, DT.91336254|
|NIA Mouse Gene Index||U042686|
|Consensus CDS Project||CCDS19651.1, CCDS19652.1, CCDS19653.1, CCDS51644.1|
|International Mouse Phenotyping Consortium Status||P2rx7|
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Tumor Biology (MTB), Gene Ontology (GO), MouseCyc
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