|
Reference
Figure
ID
|
J:85738
MacLean HE, et al., Dev Biol. 2003 Oct 1;262(1):51-63
3
MGI:5505282
|
|||||||||||||||||||||||||||||||||||||||||||||
| Image |
|
|||||||||||||||||||||||||||||||||||||||||||||
| Caption | In situ hybridization performed on tibial sections from wild-type and c-maf-null littermates. HC, hypertrophic chondrocytes; PS, primary spongiosa. | |||||||||||||||||||||||||||||||||||||||||||||
| Copyright | Reprinted with permission from Elsevier from 10.1016/S0012-1606(03)00324-5 Dev Biol 262: 51-63, MacLean HE; Kim JI; Glimcher MJ; Wang J; Kronenberg HM; Glimcher LH, Absence of transcription factor c-maf causes abnormal terminal differentiation of hypertrophic chondrocytes during endochondral bone development. Copyright 2003 | |||||||||||||||||||||||||||||||||||||||||||||
|
Associated Assays |
|
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB)), Gene Ontology (GO) |
||
|
Citing These Resources Funding Information Warranty Disclaimer, Privacy Notice, Licensing, & Copyright Send questions and comments to User Support. |
last database update 12/30/2025 MGI 6.24 |
|
|
|
||