Reference
Figure
ID
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J:85738
MacLean HE, et al., Dev Biol. 2003 Oct 1;262(1):51-63
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MGI:5505281
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Caption | Tibial sections from E14.5-E18.5 embryos and postnatal day 24 mice. (A) Bright field (BF) and (B) dark field images of sections, with hypertrophic chondrocytes (HC) and primary spongiosa (PS) bracketed. Perichondrium (P, arrow). (C,D) In situ hybridization. (E) Control in situ hybridization using sense probe in E16.5 wild-type and anti-sense probe in E16.5 null (-/-) tibia. Original magnification, 10X. | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
Copyright | Reprinted with permission from Elsevier from 10.1016/S0012-1606(03)00324-5 Dev Biol 262: 51-63, MacLean HE; Kim JI; Glimcher MJ; Wang J; Kronenberg HM; Glimcher LH, Absence of transcription factor c-maf causes abnormal terminal differentiation of hypertrophic chondrocytes during endochondral bone development. Copyright 2003 | ||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
Associated Assays |
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Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB)), Gene Ontology (GO) |
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