of Mice: CPB
Inbr: 42. Genet: b
Origin: At Centraal Proefdierenbedrijf
TNO, Holland (Cpb), about 1950.
Females often have a pair of non-lactating supernumerary nipples (Staats,
Inbr: 54. Genet: a, B, c, d, P.
Origin: As CPB-H. Former designation
Excitable; small litters; amylase type B (other CPB strains are type A).
Inbr: 37. Genet: c
. Origin: Institute of Tropical Hygiene, Amsterdam,
to Cpb 1957.
100% brachypodism due to a recessive gene.
Inbr: 47. Genet: a
, b, p.
Origin: As CPB-H.
Normal sex difference in major urinary protein content of serum and in
reaction to hexobarbital.
Inbr: 39. Genet: a, b, d, p.
Reacts to LSD with head twitching. Very light red-eyed grey.
Inbr (Cpb): ? + 61. Genet: a, B, ce, D,
Origin: Hagedoorn, Holland, to Hirschfeld 1937, to Cpb 1949.
More susceptible to tuberculosis than most strains; reacts to LSD with
Inbr: ?+50. Genet: a, b, c, D, P.
Origin: `Rhodesfarm', South
Africa, to Compton, England, to Hagedoorn, Holland, to Cpb 1949.
Inbr: ? + 59. Genet: A, B, c, D, P.
Origin: Rockefeller Institute,
probably same origin as BSVS, BRVR, etc., to Laidlaw (Hampstead), to Hagedoorn,
to Cpb 1949.
Rather aggressive, especially males (Cpb), territorial, with a sensitive
aggression-flight balance; adapted to surface-living rather than hole-living.
Inbr: 37. Genet: a, b, d, s, W.
Origin: Cross between CPB-T and
CPB-K in 1953.
Inbr: 54. Genet: cch
Hagedoorn, to Cpb 1949.
Males have low major urinary protein in serum; no sex difference in response
Inbr: 33. Genet: a
. Origin: Waltzing mice from Hagedoorn in 1949.
Crossed with strain G, now extinct, in 1953.
Carries a waltzer-shaker-like mutant; expression varies from fairly normal
walking to circling; very excitable; not deaf.
Inbr: 39. Genet: Wv/w, A.
Gruneberg, to Cpb in 1956.
Staats J. (1976) Standardized
nomenclature for inbred strains of mice: Sixth listing. Cancer Res.
INBRED STRAINS OF MICE
Updated 9 Apr. 1998
MRC Toxicology Unit, Hodgkin Building,
University of Leicester,