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Marker Summary for Reference
Reference J:144553
He H, et al., Pot1b deletion and telomerase haploinsufficiency in mice initiate an ATR-dependent DNA damage response and elicit phenotypes resembling dyskeratosis congenita. Mol Cell Biol. 2009 Jan;29(1):229-40
 
 

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last database update
04/30/2024
MGI 6.23
The Jackson Laboratory