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Gene Expression Data
Assay Details
Assay
Reference: J:118119 Sakaki-Yumoto M, et al., The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development. Development. 2006 Aug;133(15):3005-13
Assay type: RNA in situ
MGI Accession ID: MGI:5792913
Gene symbol: Sall4
Gene name: spalt like transcription factor 4
Results
Specimen 1D +/+: embryonic day 5.8 (more )
Structure Level Pattern Image Note
TS8: epiblast Present 1D Sall4 +/+
TS8: extraembryonic ectoderm Weak 1D Sall4 +/+
TS8: primitive endoderm Weak 1D Sall4 +/+
Specimen 1D -/-: embryonic day 5.8; Sall4tm1Ryn/Sall4tm1Ryn (more )
Structure Level Pattern Image Note
TS8: epiblast Absent 1D Sall4 -/-
TS8: extraembryonic ectoderm Absent 1D Sall4 -/-
TS8: primitive endoderm Absent 1D Sall4 -/-
Specimen 5C: embryonic day 8.5 (more )
Structure Level Pattern Image Note
TS14: embryo mesenchyme Present Regionally restricted 5C Sall4 Expression was detected in the mesenchyme of the anterior portion.
TS14: tail bud Present 5C Sall4
Contributing Projects:
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB)), Gene Ontology (GO)
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03/25/2025
MGI 6.24 		The Jackson Laboratory