Homozygotes for null or hypomorphic alleles do not respond to cyclic compounds (e.g., dioxin) and are resistant to their teratogenic effects. Depending on the allele, null mutants may also have liver defects, impaired female fertility, neonatal or postnatal lethality, and spleen abnormalities.
|VEGA Gene Model||OTTMUSG00000036999 (Evidence)|
|Ensembl Gene Model||ENSMUSG00000019256 (Evidence)|
|Entrez Gene||11622 (Evidence)|
|DFCI||TC1602896, TC1618782, TC1740581|
|DoTS||DT.101125370, DT.101135033, DT.101353216, DT.94402283|
|NIA Mouse Gene Index||U013861|
|Consensus CDS Project||CCDS36434.1|
|International Mouse Phenotyping Consortium Status||Ahr|
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Tumor Biology (MTB), Gene Ontology (GO), MouseCyc
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