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nitric oxide synthase 2, inducible
Phenotype annotations related to homeostasis/metabolism
*Aspects of the system are reported to show a normal phenotype.
Darker colors indicate more annotations

Mouse Phenotypes
homeostasis/metabolism phenotype
increased myocardial infarction size
abnormal platelet physiology
abnormal platelet aggregation
abnormal homeostasis
increased circulating creatinine level
abnormal blood homeostasis
abnormal circulating glucose level
increased circulating antidiuretic hormone level
abnormal circulating estradiol level
increased circulating prolactin level
decreased circulating insulin level
decreased circulating leptin level
increased circulating ketone body level
increased circulating cholesterol level
increased circulating triglyceride level
increased circulating VLDL triglyceride level
decreased circulating alanine transaminase level
decreased circulating aspartate transaminase level
abnormal circulating serum albumin level
abnormal blood coagulation
abnormal enzyme/coenzyme level
abnormal nitric oxide homeostasis
abnormal glucose homeostasis
improved glucose tolerance
abnormal glycogen homeostasis
decreased liver glycogen level
increased urine glucose level
increased insulin sensitivity
abnormal hormone level
decreased adiponectin level
abnormal adrenocorticotropin level
increased liver triglyceride level
increased triglyceride level
increased urine pH
decreased urine prostaglandin level
increased renin activity
increased mortality induced by ionizing radiation
abnormal bone healing
delayed wound healing
enhanced wound healing
decreased cerebral infarction size
decreased susceptibility to kidney reperfusion injury
decreased susceptibility to injury
Availability Mouse Genotype
Nos2tm1Lau/Nos2tm1Lau *
Nos2tm1Mrl/Nos2tm1Mrl *

Contributing Projects:
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB), Gene Ontology (GO)
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last database update
MGI 6.14
The Jackson Laboratory